Lenda VET CAT Cardiac

• Format 2 - 10Kg.
• Limited sodium content
• Renal function support
• Probiotics to support renal and digestive function.
• Adapted content of EPA and DHA fatty acids to support the cardiac muscle
• PNP 24

Ingredients

Fish (white fish hydrolysate, fresh Salmon* 10%), sweet Potato*, pulses (Lentils* min. 5%), poultry hydrolysate, Potato*, Chicken fat, Brewer's yeast (Saccharomyces cerevisiae), lignocellulose, Salmon oil, dehydrated Krill (Euphausia superba, source of Omega-3 fatty acids and astaxanthin), carob meal*, FAEC Complete (glucosamine, chondroitin sulphate, methylsulfonylmethane, Yucca schidigera extract, yeast products,(Saccharomyces cerevisiae, as a source of MOS and nucleotides), Chicory root, Salvia rosmarinus, Melissa officinalis, Taraxacum officinale, Salvia officinalis, Mentha, Minthostachys verticillata, Cynara scolymus, Silybum marianum), FAEC Inmune (microalgae oil Schizochytrium sp. (source of Omega 3 DHA fatty acids), Panax ginseng, Punica granatum, Zingiber officinale, Urtica dioica, Camellia sinensis, Curcuma longa, Harpagophytum procumbens), dehydrated vegetables (Carrot, Pumpkin...), Apple, dehydrated whole Egg, FAEC ProbioDigestive (Enterococcus faecium, Lactobacillus plantarum, Lactobacillus acidophilus, Lactobacillus rhamnosus).-*Natural raw materials.

Analytical constituents

Crude Protein 25%, crude Fat 20%, crude Ash 6.5%, crude Fibre 4.5%, Moisture 7%, Calcium 0.8%, Phosphorus 0.5%, Potassium 0.8%, Sodium 0.2%, Mg <0.1%, DHA+EPA 0.49%, ME 3995 Kcal/Kg.

Helps heart function in cases of chronic heart failure.

Heart disease in cats includes a wide range of diseases of varying severity and origin; congenital heart disease such as stenosis, dysplasia and other less common malformations may occur. Another cause would be acquired heart disease, see dilated cardiomyopathy, hypertrophic cardiomyopathy, mitral insufficiency, but whatever their etiology, they can lead to heart failure, which is broadly speaking, a dysfunction of the heart due to a structural or functional alteration of the same.

The main function of the heart is to maintain stable blood pressure and cardiac output, defining output as the volume of blood pumped by a ventricle per minute. If cardiac output decreases, it becomes impossible to maintain normal blood pressure, the pressure decreases, which triggers a neurohormonal reaction that increases heart rate, sodium and water retention, and an increase in vasoconstriction in order to compensate for this drop. This mechanism, which is effective in an acute case, is harmful in cases of cardiac dysfunction.

The most important thing in clinical treatment is to differentiate between chronic HF or acute HF, being imperative to control the acute and reorient towards a chronic treatment, which focuses on prolonging life expectancy and alleviating or minimizing symptoms.
Depending on the symptoms, the different types of HF can be identified. If we observe fluid in the abdominal cavity, we will establish that the HF affects the right side of the heart; If we observe fluid or edema in the lungs, we will establish a congestive HF, its monitoring and medication being essential, seeking the greatest possible inactivation and relaxation, forced oxygenation and the use of intravenous diuretics, in case of fluid in the abdomen, the treatment will be similar, with the exception that this type of congestion will not cause asphyxia.

The veterinary professional, based on the evaluation and diagnostic tests, will establish the appropriate pharmacological treatment, with diuretics, anticoagulants, inhibitors... Nutritional treatment being an important factor in the treatment of this syndrome.

Focusing on the nutritional level, help for cardiac function is given by various aspects as varied as mineralization, supplementation with fatty acids, caloric control...
The correct diet for the treatment of this syndrome should promote normal weight, avoiding overweight that generates an increase in cardiac hemodynamic demand, predisposing the appearance of arrhythmias and will interfere with normal breathing.

On the other hand, cachexia must be prevented, so it must be a tasty recipe that encourages its consumption, calorically adjusted since caloric restriction suggests a reduction in oxidative stress and excess would promote overweight, with a balanced level of protein, which limits protein catabolism that would affect the myocardium. Sadly, the detection of cachexia is a symptom of an advanced state of HF.

It is considered that supplementation with Taurine and L-Carnitine is not of major relevance in specimens in which there is no Taurine deficiency, and its supplementation is also recommended in specimens with hypokalemia derived from kidney disease.

Another especially useful factor that will distinguish an adequate diet is the use of Omega-3 fatty acids; these long-chain fatty acids, rich in EPA and DHA, contribute to minimizing the appearance of cachexia. There is a possibility that the anti-inflammatory action of these fatty acids may be equally beneficial in cats, as it is in humans and dogs, although there are no studies on this yet.
Logically, the correction of levels of Potassium, Magnesium and Sodium and even Phosphorus should be taken into account.

The relevance of antioxidants in the management of heart disease is fundamental, as are vitamins E and C, botanical extracts as a source of flavonoids, Taurine itself... Having demonstrated the benefits of probiotics in other species to attenuate heart failure and as there are still no studies in cats, we will focus on their beneficial effect in any pharmacological treatment or on their fundamental immunomodulatory effect.